If you know this chemical ingredient of astaxanthin, then you want to know more about astaxanthin, this is definitely an article that can help you.
By citing the eight benefits of astaxanthin to the human body, you can help you learn more about this chemical materials.
The human retina and the central nervous system (brain) are rich in unsaturated fatty acids, so free radicals generated by oxidation can easily cause peroxidative damage. Studies have shown that astaxanthin easily passes through the blood-brain barrier and cell membrane, which can effectively prevent retinal oxidation and photoreceptor cell damage, as well as protect the central nervous system, especially the brain, so as to effectively treat ischemia. Central nervous system damage such as perfusion injury, spinal cord injury, Parkinson's syndrome, Alzheimer syndrome. In particular, the effect of macular degeneration is more pronounced than lutein. American CADAX has used astaxanthin as a drug to prevent re-infarction after cerebral infarction, and it has been reported that it has entered clinical trials in 2010. The biggest benefit is that it does not cause coagulopathy like antiplatelet drugs.
As we all know, ultraviolet radiation is an important cause of skin aging and skin cancer. Studies have shown that astaxanthin has a special effect on transglutaminase, which can consume putrescine when the skin is exposed to light. Oral astaxanthin has a stronger inhibitory effect on the accumulation of putrescine than oral vitamin A. Therefore, the strong antioxidant properties of astaxanthin may make it a potential photoprotective agent, effectively remove free radicals that cause skin aging, protect cell membranes and mitochondrial membranes from oxidative damage, and prevent skin aging.
Clinical studies of arteriosclerosis and related cardiovascular diseases have shown that oxidation of low-density lipoprotein (LDL) is an important cause of arteriosclerosis. The higher the concentration of LDL in the human body, and the thinning of blood vessels caused by platelet deposition, the greater the risk of blood flow, and the greater the risk of arteriosclerosis in the human body. High-density lipoprotein (HDL) in the blood has the opposite effect, which prevents arteriosclerosis. In human blood, astaxanthin is carried by very low density lipoprotein (VLDL), LDL and HDL. Clinical trials have shown that oral administration of 3.6 mg/d astaxanthin per person for 2 weeks can prevent oxidation of LDL. In addition, animal studies have shown that astaxanthin has a significant increase in HDL and LDL reduction in vivo, and HDL can be increased from the original 49.7 ± 3.6mg / dL to 66.5 ± 5.1mg / dL, so It is speculated that astaxanthin can reduce the oxidation of apolipoprotein and can be used to prevent arteriosclerosis, coronary heart disease and ischemic brain damage. Unlike the drugs that generally lower LDL, the LDL oxidation time was prolonged by 5.0%, 26.2%, 42.3%, and 30.7%, respectively, after taking 1.8mg, 3.6mg, 14.4mg, and 21.6mg astaxanthin for 2 weeks. Thereby preventing the occurrence of atherosclerosis.
Astaxanthin mainly passes:
Increase high density lipoprotein HDL-C
Reduce LDL oxidation to Ox-LDL
Reduce the inflammatory response of macrophages
Reduce the formation of atherosclerotic plaque
Increase the stability of atherosclerotic plaque
Reduce plaque rupture and improve blood flow
The above methods are used to prevent cardiovascular diseases.
Astaxanthin can significantly affect the immune function of animals. In the presence of antigen, it can significantly promote the ability of spleen cells to produce antibodies, enhance the role of T cells, and stimulate the production of immunoglobulin in vivo. Astaxanthin can also partially restore the humoral immune system of aged mice, which can increase the IgM, IgA and IgG in mice to 10mol/L, respectively, indicating that it can enhance specific humoral immune response in the early stage of antigen invasion. . In addition, astaxanthin can also enhance the ability of mice to release interleukin-Iα and tumor necrosis factor alpha, which is much stronger than β-carotene and canthaxanthin. Therefore, astaxanthin has a strong activity of inducing cell division and has an important immunomodulatory effect.
When the body moves, the muscles release free radicals. If these free radicals are not treated by the antioxidants in time, they will produce oxidative stress, causing muscle soreness or damage to muscle tissue. Studies have shown that astaxanthin can act as an antioxidant to inhibit the oxidative damage of free radicals to the body. In addition, oral astaxanthin can also strengthen aerobic metabolism, increase muscle strength and muscle tolerance, quickly relieve exercise fatigue, and reduce delayed muscle pain caused by strenuous exercise. The main performance is: 4mg / day, after 6 months can increase body strength by 40%. 4mg/day, after 2 weeks, can extend the duration of continuous exercise by 20% and reduce the accumulation of lactic acid after exercise by 28.6%.
Joint pain and arthritis are usually caused by oxidative damage caused by free radicals. The strong antioxidant properties of astaxanthin help to inhibit free radicals and reduce their oxidative damage to joints. Studies have shown that feeding mice with astaxanthin-rich Haematococcus pluvialis can activate the response of T lymphocytes, thereby reducing the adhesion and infection of Helicobacter pylori to the stomach. Mara studied the effects of Haematococcus astaxanthin products on human health and compared it with 26 other well-known anti-inflammatory drugs. The results showed that the health status of patients taking astaxanthin increased by 85%, astaxanthin and 92% of anti-inflammatory drugs have the same effect or better results. Studies have shown that 4mg/day astaxanthin inhibits inflammatory factors (such as prostaglandin E) and is equivalent to 4mg (cortisone), but it has no side effects (cortisone), so it is also known as a hormone without side effects.
Studies by Tanaka et al. showed that feeding rats and mice with astaxanthin 100-500 mg/kg significantly inhibited chemical-induced initial carcinogenesis, and had anti-proliferative effects and enhanced immunity against epithelial cells exposed to carcinogens. The role of function, and this effect has a dose-effect relationship. Compared with the control group, the tumor incidence and tumor size in the high dose group (500 mg/kg) were significantly lower than those in the control group and the low dose group. Therefore, it is speculated that astaxanthin has significant anticancer properties. Astaxanthin can also induce transferase in the liver, significantly inhibiting mouse bladder cancer, rat oral cancer, colon cancer, and gastric cancer, and its effect is more obvious than that of β-carotene. In addition, astaxanthin can also prevent the carcinogenicity of aflatoxin, and has a good effect on reducing the amount and volume of aflatoxin-induced liver tumor cells.
Researchers have proposed the anti-cancer mechanism of astaxanthin, which is thought to be related to cell membrane stability and protein gene expression. It regulates cell-to-cell communication by changing membrane stability and gene expression, thereby improving cell-to-cell balance and maintaining cells. The normal function.
70% of people with diabetes develop nephropathy within 5 years. Astaxanthin is the only substance that can effectively prevent diabetic nephropathy. Astaxanthin is mainly destroyed by directly protecting the glomerular basement membrane and preventing free radicals produced by hyperglycemia. Basement membrane. In addition, it can also fight free radicals in renal tubular epithelial cells, protect the normal transport of glucose and phosphorus in renal tubular cells, thereby preserving important substances such as ATP and sodium-potassium ATPase, ensuring that blood flow in the kidney is not affected, reducing proteinuria. The production.
Studies have confirmed that 8 mg of astaxanthin can significantly reduce urine protein by 70% in 8 weeks.
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